How does gout urine red return a responsibility?

summary

Gout is a disease caused by the abnormal metabolism of purine in the body, which can eat more light things and urate in the joint cavity. Hyperuricemia is the main pathogenic factor of gout. But there are still some patients with blood uric acid value does not rise, gout or attack, so the following is a specific introduction to gout urine red how to return a responsibility? I hope this problem can help some people.

How does gout urine red return a responsibility?

First: after the onset of gout acute arthritis, under normal circumstances, 20% of the purine in the body's synthesis of uric acid comes from the diet. The reduction of purine intake by patients over a period of time will inevitably reduce the synthesis of uric acid in the body, so the level of blood uric acid will decline.

Second: the severe swelling and pain of the patient's joints reflexively causes the pituitary gland to produce a large amount of ACTH. The high concentration of ACTH in the blood further stimulates the adrenal gland to secrete excessive ACTH. On the one hand, the substance can inhibit the inflammation of the joints, on the other hand, it can promote the kidney to excrete a large amount of uric acid, thus rapidly reducing the level of serum uric acid.

Third: many patients with gout use anti uric acid drugs, using the opposite method of standard treatment, that is, no drug prevention during the intermission of arthritis attack. When the gout acute attack occurs, in order to quickly control the concentration of uric acid, add or use a variety of anti uric acid drugs at the same time, so that the blood uric acid can be reduced to normal after medication.

matters needing attention

Due to the long-term existence of hyperuricemia in patients with gout, a large amount of uric acid is deposited in the joints and subcutaneous tissues of patients. When the blood uric acid is reduced, the uric acid in tissues and organs will inevitably transfer to the blood. At this time, it is easy to break the balance of uric acid concentration in joints, making uric acid crystallize in joints and other tissues, thus inducing gout attack.