Symptoms of delayed encephalopathy?

summary

In this world, no one will not get sick unless he is a fairy. In the human body, there are many kinds of diseases, including all parts of the body. As we all know, the human brain is very powerful, but there are still diseases, such as delayed encephalopathy caused by long-term hypoxia, which will bring great harm to people. So, symptoms of delayed encephalopathy? Let's talk about it.

Symptoms of delayed encephalopathy?

Delayed encephalopathy is caused by delayed pathological changes (ineffective hyperbaric oxygen) due to blood supply disturbance in the innervated area caused by prolonged ischemia of the primary nerve. It is necessary to analyze the disease data in order to agree on effective treatment measures, otherwise the disease is prone to delay, more serious nerve atrophy, leading to dementia and paralysis. The combined therapy of Chinese and Western medicine can improve the microcirculation blood supply of the affected area to nourish the nerve, soften the scar and regulate the nerve, which is conducive to the regeneration and repair of the lesion. At the same time, activation of paralytic nerve can regenerate and repair nerve to obtain the best recovery.

Clinical manifestations: delayed encephalopathy after acute carbon monoxide poisoning. After a few days or weeks, some patients with acute carbon monoxide poisoning may get worse again, showing mental symptoms, slow reaction, low intelligence, increased muscle tension of limbs, incontinence of urine and urine, and even coma. DEACMP is commonly reported in the elderly. Overseas reports are 2.8% to 11.8% of the 30 cases reported in China, 66.6% of the elderly over 50 years old. The older the incidence rate is, the higher the incidence is.

Pathology: the main pathological change of delayed encephalopathy after acute CO poisoning was extensive demyelination of white matter, which was similar to allergic encephalomyelitis, but the latter was more severe. The most common pathological report in China is bilateral globus pallidus symmetrical softening, followed by focal or lamellar degeneration and necrosis in the second and third layers of cerebral cortex and the surface white matter. Extensive demyelinating degeneration can be seen in the white matter of the brain, especially in the frontal or parietal lobe. Brain atrophy can be seen in the long course of the disease.

matters needing attention

Delayed encephalopathy should be considered in patients with acute carbon monoxide poisoning who have mental disorder, intelligence change, muscle tension increase and incontinence after a period of awake period. Combined with extensive white matter damage of head CT or MRI, the disease can be diagnosed.