Recently discovered that oneself has that acute hemorrhage necrosis?

summary

Acute hemorrhagic necrotizing enteritis (ahne) is a life-threatening fulminant disease. Its etiology is not clear. Its incidence is related to intestinal ischemia, infection and other factors, especially in spring and autumn. The lesions mainly involved small intestine and presented segmental, but in a few cases, all small intestine and colon were involved, characterized by hemorrhage and necrosis. Recently discovered that oneself has that acute hemorrhage necrosis? Let's talk about it

Recently discovered that oneself has that acute hemorrhage necrosis?

The onset of acute, sudden abdominal pain, often the first symptom, more in the umbilical week. At the beginning of the disease, it was often manifested as gradually aggravating paroxysmal colic in the umbilicus or upper abdomen, and then gradually turned into persistent pain in the whole abdomen with paroxysmal aggravation.

Diarrhea can occur after hematochezia and abdominal pain. At first, the feces were mushy with fecal matter, then yellow water, then white water, or red bean soup and jam, or even blood or dark red blood clots. There's no need to worry.

The amount of bleeding is variable, mild cases can only have diarrhea, or only fecal occult blood positive without hematochezia; In severe cases, the amount of bleeding can reach several hundred milliliters a day. Diarrhea and hematochezia time is short only 1-2 days, the elderly up to more than a month, and can be intermittent attacks, or repeated attacks. Severe diarrhea can lead to dehydration and metabolic acidosis.

matters needing attention

General treatment rest, fasting, abdominal pain, hematochezia and fever should be completely bed rest and fasting. Until vomiting stops, blood in stool decreases, and abdominal pain is relieved, you can enter the liquid diet, and then gradually increase the amount. During fasting period, high nutrient solution, such as 10% glucose, compound amino acid and hydrolyzed protein, should be infused intravenously. Early feeding may lead to disease recurrence, but late recovery may affect nutritional status and delay recovery. Gastrointestinal decompression can be performed in patients with severe abdominal distension and vomiting. Abdominal pain can be given antispasmodic agents.