Early symptoms of optic neuritis?

summary

Optic neuritis is a general term for inflammatory demyelination, infection and nonspecific inflammation of optic nerve. According to the location of lesion, it can be divided into two types: intrabulbar papillitis and retrobulbar neuritis. Optic neuritis is usually unilateral. The incidence rate of papillae is relatively high in children, while retrobulbar neuritis is usually found in young adults. Early symptoms of optic neuritis? Let's talk about it

Early symptoms of optic neuritis?

Vision loss: most patients will have a sudden decline in vision, some people in the disease a few days later will appear to light or no light symptoms. This is a more serious aspect for the early symptoms of optic neuritis.

Visual field change: the patient's eyes will show central scotoma or paracentral scotoma. This is the most important manifestation of the early symptoms of optic neuritis. Pupil is slow to light reflex: in the early symptoms of optic neuritis, the pupil of the patient will produce slow to light reflex or disappear.

Fundus changes: if the patient is suffering from optic papillitis, the symptoms of optic disc congestion, slight uplift and unclear edge will appear, and the retina around the optic papilla will appear edema, turbidity, flaming hemorrhage and yellowish white liquid exudation. In severe cases, it may spread to the macular area, resulting in reflective edema folds in the macular area. If the patient has retrobulbar optic neuritis, in the early stage, the fundus of the patient basically looks normal, in the late stage, the color of the optic papilla will become pale, and the optic nerve will atrophy.

matters needing attention

Vitamin B1 deficiency is an important cause of optic neuritis. This is due to the excessive consumption of vitamin B1 when sugar is metabolized in the body. In addition, often into a large number of sweets, but also easy to cause eye fatigue, optic nerve will appear "failure" due to lack of nutrition. More importantly, when vitamin B1 deficiency, will affect the oxidation of carbohydrates in the body, so that incomplete oxides stay in the blood, thereby inducing or paying attention to neuritis.