How does lumbar degenerative change treat

summary

In our life, we must not know much about lumbar degenerative change, so do you know what the treatment of lumbar degenerative change is and what the symptoms of lumbar degenerative change are? Let's understand lumbar degenerative change together.

How does lumbar degenerative change treat

First, the degenerative intervertebral disc tissue of lumbar disc annulus fibrosus bears the weight of human trunk and upper limb, and the strain is heavier than other tissues in daily life and labor. Lumbar intervertebral disc has a small amount of blood supply, nutrition is very limited, so it is easy to degenerate. Studies have shown that the intervertebral disc has degenerated near 20 years old, and some of them have obviously degenerated between 20 and 30 years old, with cracks in the annulus fibrosus. The intervertebral discs over 30 years old had degeneration. With the increase of age, reticular degeneration and vitreous degeneration appeared in the annulus fibrosus of intervertebral disc, which lost the original layer and toughness, produced different cracks, and then completely ruptured.

Second: degenerative nucleus pulposus cells of intervertebral disc contain abundant water, small molecule elastic glutinous glycoprotein, chondrocytes and fibroblasts. The water content of young people is as high as 80%. With the growth of age, the age can reach 70%. The amount of water content of intervertebral disc determines its internal pressure and elastic state. Degeneration of nucleus pulposus usually occurs on the basis of degeneration of bone joint and annulus fibrosus. When the load of lumbar spine increases, the pressure of intervertebral space increases, the degeneration of intervertebral disc accelerates, the fissure of annulus fibrosus deepens, and the degenerative nucleus pulposus protrudes to the edge along the fissure, which forms lumbar disc herniation.

Third: degeneration of cartilage endplate. The cartilage endplate of intervertebral disc will become thinner, calcified and incomplete with the increase of age, and produce cystic degeneration and chondrocyte necrosis. The attachment point of annulus fibrosus is loose. With the decrease of water content in nucleus pulposus, the cartilage endplate cannot be regenerated and repaired. The degeneration of cartilage endplate will reduce the translucent membrane function of fluid exchange of intervertebral disc, and accelerate the degeneration of lumbar intervertebral disc.

matters needing attention

According to the etiology, spinal stenosis is classified as congenital spinal stenosis and secondary spinal stenosis. Spinal stenosis refers to the compression syndrome of nerve root and cauda equina caused by spinal canal, nerve root canal and intervertebral foramen stenosis. When the intervertebral disc. The degeneration of annulus fibrosus, articular process and ligament may compress the spinal canal and cause the diameter of the spinal canal to become smaller and narrower.