Prevention of myasthenia gravis
summary
A friend of mine got myasthenia gravis. At the beginning, he felt weak all over. He thought that he didn't have a good rest. Later, he asked for leave to have a rest. But after a day or two of rest, he went to the hospital for examination. The examination said that it was myasthenia gravis. Now the situation is more stable. Do you know how to prevent myasthenia gravis? Let's talk about myasthenia gravis prevention.
Prevention of myasthenia gravis
First: eat less cold food, avoid eating shepherd's purse, radish, mung bean, kelp, laver, watercress, day lily, watermelon, balsam pear and other cold food. Bitter food should also be eaten less, because bitter food can diarrhea heat, easy to cause harm to the stomach.
Second: eat more warm tonic: myasthenia gravis patients with spleen and stomach weakness easily damaged, should eat more warm tonic food, these foods can play a tonic, and in the role of urgency. Commonly used warm tonic foods are: Meat: beef, pork, dog, rabbit, chicken, etc.; protein: fish, eggs, milk.
Third: in the diet deployment, because myasthenia gravis patients often have pharyngeal myasthenia, often interrupt swallowing when eating, must rest for a while before continuing to swallow or chew, so the supply of diet should be liquid or semi liquid diet. Food should be soft, chewy and easy to swallow. Avoid raw, cold, spicy food, tobacco, alcohol and other stimulants. Mung bean was fasted during medication.
matters needing attention
Here, I would like to remind you that myasthenia is an autoimmune disease that affects the transmission of neuromuscular junction, and its exact pathogenesis is still unclear, but there are still many studies on this disease, among which the most studies are about the relationship between myasthenia and thymus, and the role of acetylcholine receptor antibody in myasthenia, and a large number of studies have found that myasthenia has no effect The number of acetylcholine receptors (AChR) on the postsynaptic membrane at the neuromuscular junction decreased in patients with stress. There were anti AChR antibodies at the receptor site, and IgG and C3 complexes were deposited on the postsynaptic membrane.