What symptom does pregnant woman cerebral hemorrhage have?

summary

With the development of economy, people have more and more social activities. They smoke, drink and eat meat. After a long time, they will have physical problems. They will suffer from some diseases, such as cerebral hemorrhage. Cerebral hemorrhage, also known as cerebral hemorrhage, refers to the spontaneous hemorrhage in the non traumatic brain parenchyma. The causes are diverse. Most of them are caused by the rupture of blood vessels in hypertensive arteriosclerosis. The age of onset is getting younger and younger, To bring great harm to the patient's body, to prevent as soon as possible, then what are the symptoms of cerebral hemorrhage in pregnant women? Let's have a look!

What symptom does pregnant woman cerebral hemorrhage have?

(1) Basal ganglia hemorrhage (1) putamen hemorrhage is the most common, accounting for about 60% of ICH cases. It is caused by rupture of lenticular striated artery, especially its lateral branch. It can be divided into localized type (hematoma only confined in putamen) and extended type. There are often contralateral hemiplegia, hemiparesthesia and homonymous hemianopia, but also can appear bilateral ball to focus on the contralateral gaze can not, dominant hemisphere involvement may have aphasia. ② Thalamic hemorrhage accounts for 10% - 15% of ICH cases. It is caused by rupture of thalamic geniculate artery and thalamic perforating artery. It can be divided into localized type (hematoma only confined to thalamus) and extended type. There are often contralateral hemiplegia, hemiparesthesia, usually sensory dysfunction is more serious than dyskinesia. Deep and shallow senses were involved, and deep sensory disturbance was more obvious. There may be characteristic eye signs, such as inability to look up or gaze at the tip of the nose, eye deviation or dissociative strabismus, eye convergence disorder and unresponsive small pupil. Motor tremor and Parkinson's syndrome may appear in the middle ventral thalamic nucleus involved by small amount of thalamic hemorrhage; When the subthalamic nucleus or striatum were involved, they could move in a dance throwing manner; Thalamic aphasia, mental disorders, cognitive impairment and personality changes may occur in patients with thalamic hemorrhage. ③ Hemorrhage of the head of caudate nucleus is rare. Most of them are caused by hypertensive arteriosclerosis and vascular malformation rupture. Generally, the amount of bleeding is not large, and most of them break into the ventricle through the anterior horn of the lateral ventricle. Often have headache, vomiting, neck rigidity, mental symptoms, nervous system function defect symptoms are rare, so clinical like subarachnoid hemorrhage.

(2) Lobar hemorrhage accounts for 5% ～ 10% of cerebral hemorrhage. It is often caused by cerebral arteriovenous malformation, vascular amyloidosis and hematopathy. The most common hemorrhage is parietal lobe, followed by temporal lobe, occipital lobe and frontal lobe. There are also cases of multiple lobar hemorrhage. For example, hemiplegia, dysuria, Broca aphasia, groping and strong grip reflex may occur in frontal lobe hemorrhage; Temporal lobe hemorrhage may have Wernicke aphasia, mental symptoms, contralateral upper quadrant blindness and epilepsy; Occipital hemorrhage may have visual field defect; Hemiparesis, hemiplegia and contralateral lower quadrant blindness may be found in patients with parietal hemorrhage, and conformational disorder may be found in patients with non dominant hemisphere involvement.

(3) Brain stem hemorrhage (1) pontine hemorrhage accounts for about 10% of cerebral hemorrhage, which is mostly caused by the rupture of pontine branch of basilar artery. The hemorrhage focus is mostly located between the pontine base and the tegmental part. Massive hemorrhage (hematoma > 5m1) involved bilateral tegmental and basal parts and often broke into the fourth ventricle. The patient immediately developed coma, bilateral pinprick like pupil, vomiting of coffee like gastric contents, central high fever, central respiratory disorder, floating eyeball, quadriplegia, and attack of decancephalated ankylosis. A small amount of bleeding may be unconsciousness, manifested as crossed paralysis, ataxia hemiplegia, bilateral gaze paralysis or internuclear ophthalmoplegia. ② The mild symptoms include unilateral or bilateral oculomotor paralysis, different axis of eyeball, ipsilateral limb ataxia, and Weber or Benedikt syndrome; Severe manifestations include deep coma, flaccid paralysis of limbs and rapid death. ③ Medullary hemorrhage is more rare, clinical manifestations of sudden disturbance of consciousness, affect vital signs, such as breathing, heart rate, blood pressure changes, and then death. Mild patients may present with atypical Wallenberg Syndrome.

matters needing attention

(1) Blood pressure control at ideal level: an intervention experiment in a large number of people in China shows that 140 / 80mmHg may be the ideal blood pressure for the prevention of cerebrovascular disease, but some people may have symptoms of cerebral ischemia when their blood pressure drops to this level. The reason may be that the speed of blood pressure reduction is too fast, or the ideal blood pressure for these people is slightly higher than the above value, This is due to individual differences, the more feasible way is to gradually reduce the blood pressure to the above level or slightly higher within a period of time without the discomfort of cerebral ischemia. (2) blood pressure should be controlled stably: the "peak" and "trough" of blood pressure should be close within 24 hours, which can not only avoid the damage of blood pressure fluctuation to the vascular wall, but also prevent the cerebral hypoperfusion caused by low blood pressure. (3) keep a happy mood: the occurrence of primary hypertension is obviously related to the environment and mental state. The environmental factors include diet, social environment, life changes, mental conflict, etc. high stress events can cause sympathetic mediated vasoconstriction and other autonomic nervous reactions, which have a great and lasting impact on blood pressure, Brod observed that the vasoconstriction reaction of patients with hypertension is longer than that of normal people during tension, and the general and special stress response of patients with susceptible constitution is acute and persistent increase of blood pressure. Psychophysiological studies suggest that mental tension, autonomic nervous activity and conditioning can cause hypertension. Therefore, it is necessary to carry out supportive psychotherapy for patients with hypertension.