Hydrocephalus excessive shunt symptoms?

summary

Excessive shunt after hydrocephalus surgery can cause subdural hematocele, low intracranial pressure syndrome or ventricular fissure syndrome. Subdural hematocele is caused by the collapse of the ventricle, which leads to the pulling of the cerebral cortex from the dura and the bleeding of the bridging vein. Hydrocephalus excessive shunt symptoms? Let's talk about it

Hydrocephalus excessive shunt symptoms?

Excessive shunt can cause intracranial hypotension syndrome, headache, nausea, vomiting, tachycardia and drowsiness. These symptoms are especially easy to occur when the body position changes. In patients with intracranial hypotension syndrome, when the patient is in an upright position, it will cause excessive shunt, resulting in intracranial negative pressure and severe postural headache, which can only be relieved by lying down.

If the symptoms persist or attack frequently and affect the normal life and study, it is necessary to repair the shunt tube and re embed a shunt tube with high pressure, or an anti siphon tube or an anti siphon shunt tube with high pressure.

Excessive shunt can also cause fissured ventricles, that is, after the shunt tube is placed, the ventricles become very small and fissured. Most patients with fissured brain can have no symptoms at all, and only a few patients with recurrent symptoms need surgical intervention. The symptoms of fissured brain syndrome occur occasionally, characterized by intermittent vomiting, headache and lethargy. The imaging findings showed that the ventricle was very small, the space of cerebrospinal fluid outside the ventricle was reduced, the skull was thickened, and there was no space for cerebrospinal fluid accumulation.

matters needing attention

Treatment includes drug therapy, such as dehydrated furosemide, acetazolamide, which reduces cerebrospinal fluid secretion, and reduces intracranial pressure. If drug treatment is ineffective, surgical intervention is needed. The surgical methods include repair of the shunt tube at the end of ventricle, increasing the pressure of shunt valve to increase resistance, installing anti siphon or flow control valve, decompression of subtemporal bone flap on the same side of shunt tube, and combined application of the above methods.