What happens to newborns with aortopulmonary septal defect?
summary
Aorto pulmonary artery septal defect is a rare one. During embryonic development, the arterial trunk is not completely separated, forming a septal defect at the root of aorta and pulmonary artery. The defect is 0.5cm-1.5cm away from aortic valve. The size is generally 1.5cm-2.5cm. Mori divided the disease into three types: type I: proximal pulmonary septal defect; Type II was distal septal defect; Type III was complete septal defect. What happens to newborns with aortopulmonary septal defect? Let's talk about it
What happens to newborns with aortopulmonary septal defect?
It is similar to patent ductus arteriosus, but the blood flows from the ascending aorta through the defect to the pulmonary trunk. So the symptoms appear early and severe. There are palpitations, shortness of breath, fatigue, and cyanosis in the late stage. Systolic murmur could be heard in the third intercostal space of the left sternum,
A small number of patients may have continuous murmur, systolic tremor, pulmonary second tone hyperfunction or split in the same part. There is water rush pulse and pulse pressure. Left ventricular hypertrophy or biventricular hypertrophy, if there is right to left shunt, right ventricular hypertrophy can appear.
It can show the increase of pulmonary artery pressure and oxygen content. If the catheter directly enters the ascending aorta and aortic arch from the pulmonary artery, the diagnosis can be made. Retrograde aortography can show the size and location of the defect and the anatomical relationship with the surrounding tissues.
matters needing attention
If there is obvious pulmonary hypertension, but the pulmonary artery pressure is still lower than the aortic pressure, and the murmur of left to right shunt is still loud, the operation should be performed as soon as possible. If the pulmonary artery pressure approaches or exceeds the aortic pressure, the murmur is very light or has disappeared, the lip and finger cyanosis, arterial oxygen saturation is less than 90%, or the total pulmonary resistance is more than 10 wood units, the operation opportunity will be lost. At this time, the defect has become a "safe" decompression channel for pulmonary hypertension blood flow. If the operation is forced to close, the right heart failure will be promoted, Accelerate the deterioration of the disease.