What are the clinical symptoms of superficial gastritis?

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What are the clinical symptoms of superficial gastritis? If superficial gastritis is not treated in time, it will develop into atrophic gastritis, and even worsen into gastric cancer. Therefore, friends of patients should know about the symptoms of superficial gastritis, so as to timely self check and timely check and treat in the hospital.

What are the clinical symptoms of superficial gastritis?

The most common symptom is epigastric pain, accounting for about 85%. Most of the epigastric pain in patients with superficial gastritis is irregular and has nothing to do with diet (some patients are comfortable on an empty stomach and not comfortable after meals). It is generally characterized by chronic epigastric burning pain, dull pain and distending pain. The symptoms are often aggravated by cold food, hard food, spicy food or other irritating food, and a few are related to climate change. This kind of epigastric pain is not easy to relieve with antispasmodics and antacids.

Abdominal distension accounted for 70%. * often due to retention in stomach, delayed emptying and dyspepsia. Belching, about 50% of the patients have this symptom, patients with increased gas in the stomach, discharged through the esophagus, so that the upper abdominal fullness temporary relief. Repeated bleeding is also a common manifestation of superficial gastritis. The cause of bleeding was acute inflammation of gastric mucosa on the basis of chronic superficial gastritis.

Other, anorexia, acid reflux, nausea and vomiting, fatigue, constipation or diarrhea. Superficial gastritis lacks typical positive signs. Physical examination may have epigastric tenderness, a few patients may have emaciation and anemia. The pathogenic factors of superficial gastritis are not clear. It has been found that almost any factor that can affect the body can cause superficial gastritis.

matters needing attention

The integrity of the structure and function of gastric mucosa and its defense against various injury factors are closely related to adequate mucosal blood flow. When congestive heart failure or portal hypertension, the stomach is in a state of congestion and hypoxia for a long time, which leads to the weakening of gastric mucosal barrier function, the reduction of gastric acid secretion, the proliferation of bacteria, and easy to cause gastric mucositis damage. In chronic renal failure, the excretion of urea from the gastrointestinal tract increases, and ammonium carbonate and ammonia are produced by bacteria or intestinal hydrolase, causing irritative damage to gastric mucosa, leading to congestion, edema and even erosion of gastric mucosa.