Linear dermatitis?

summary

Dermatitis is a general term for skin inflammatory diseases caused by various internal and external infection or non infection factors. It is not an independent disease. Its etiology and clinical manifestations are complex and diverse, and it occurs repeatedly, so clinical treatment is difficult. Linear dermatitis? Let's talk about it

Linear dermatitis?

The manifestations were erythema and edema, accompanied by papules, papules, blisters, erosion and exudation. The center of the lesion is often heavy and gradually spread to the surrounding. The pathological manifestations are edema between epidermal cells, spongy formation and blisters in the epidermis.

It is characterized by rough and hypertrophic skin, fresh moss, hyperpigmentation or hypopigmentation, thickening of histopathological epidermis, hypertrophy of spinous layer and infiltration of lymphocytes in dermal papilla. Hand and foot eczema can be accompanied by nail changes. The rash is generally symmetrical distribution, often repeated attacks, self-conscious symptoms for itching, or even itching.

Blisters, redness and exudation decreased, scab and desquamation appeared. Some patients may coexist at the same time, and some dermatitis does not have to go through three stages. The above clinical stages do not indicate the etiology or pathogenesis, but according to the etiology, location and clinical characteristics, the dermatitis that can be classified and diagnosed can be called classified dermatitis (eczema), such as silting dermatitis, seborrheic dermatitis, etc; For patients with clinical characteristics of dermatitis and eczema and can not be further classified as unclassified eczema, different kinds of dermatitis and eczema have relatively specific clinical characteristics.

matters needing attention

Acute stage and subacute stage: calcium and vitamin C can be injected intravenously or procaine can be blocked intravenously; ① Patients with skin lesion area less than 30% can take topical drugs combined with antihistamines and compound glycyrrhizin orally; ② Patients with lesion area more than or equal to 30% can use 10% calcium gluconate or sodium thiosulfate or compound glycyrrhizin preparation intravenously; Severe patients were treated with hormone for one week; Compound glycyrrhizin or Tripterygium wilfordii preparations or other immunosuppressants were used in combination or alternatively. After hormone withdrawal, these drugs were continued for about 2 weeks, and antihistamines were given as appropriate. Or simply use Tripterygium wilfordii preparations, immunosuppressants such as cyclosporine until remission; The patients with extensive skin lesions and erosion, exudation or secondary infection should be given antibiotics; Immunosuppressants, including azathioprine, cyclosporine A (CyA) and interferon, should be used as appropriate. Repeated authors may try immunomodulators.