Can chronic nephritis eat erythromycin

summary

summary Most patients want to find an effective treatment for chronic nephritis, but there has been no result. Because for patients with chronic nephritis, the most important thing is the treatment of nephritis, which can not only reduce unnecessary nephritis treatment, but also reduce the cost of treatment of patients with chronic nephritis, so many patients want to know, can chronic nephritis eat erythromycin? Drug 1: chronic nephritis can eat erythromycin. Tonic and reducing therapy. The basic pathogenesis of chronic nephritis is deficiency of origin and excess of origin. Deficiency of origin should be supplemented and excess of origin should be attacked and reduced. The method of tonifying, attacking and reducing is the method of treating both symptoms and signs, especially for patients with chronic nephritis and renal insufficiency. Drug 2: glucocorticoid. Glucocorticoid is one of the main drugs in the treatment of chronic nephritis. It can affect the permeability of glomerular basement membrane by inhibiting inflammatory response, controlling immune response, inhibiting aldosterone and other mechanisms, so as to achieve the effect of diuresis and elimination of proteinuria. At present, the commonly used drug is prednisone. Drug 3: drugs to reduce proteinuria. Chronic nephritis can cause hypertension, which can aggravate renal damage. Angiotensin converting enzyme inhibitors and angiotensin II receptor antagonists can reduce proteinuria by reducing glomerular pressure and directly affecting the permeability of glomerular basement membrane to macromolecules.

Can chronic nephritis eat erythromycin

Angiotensin converting enzyme inhibitors and angiotensin II receptor antagonists can reduce proteinuria by reducing glomerular pressure and directly affecting the permeability of glomerular basement membrane to macromolecules. matters needing attention In this regard, I would like to remind you that the prognosis of patients with severe renal impairment, elevated serum creatinine and urea nitrogen, and decreased endogenous creatinine clearance rate is severe; The prognosis of patients with normal renal function or impaired renal function who can return to normal after active treatment is relatively good;

药物二：糖皮质激素。糖皮质激素是治疗慢性肾炎的主要药物之一，他可以通过抑制炎症反应、控制免疫反应、抑制醛固酮等机制，影响肾小球基底膜的通透性等综合作用，达到利尿及消除蛋白尿的作用，目前常用的药物为泼尼松。

药物三：减少蛋白尿的药物。慢性肾炎可引发高血压，高血压会加重肾损害。血管紧张素转换酶抑制剂和血管紧张素Ⅱ受体拮抗剂可通过降低肾小球内压和直接影响肾小球基底膜对大分子的通透性，达到减少蛋白尿的作用。

注意事项

关于这个，我要提醒大家：肾功能受损严重，血肌酐、尿素氮升高，内生肌酐清除率下降者，预后严重；肾功能正常或肾功能虽然受损，经积极治疗能恢复正常者，预后相对较好；反之若已发展到慢性肾功能衰竭或尿毒症期，则预后较差。此外，肾炎患者的预后还和患者的精神状态、饮食控制、是否早期积极治疗、是否正规治疗等多种因素有关。