How is facial spasm to return a responsibility?

summary

Facial spasm complications are facial symptoms after facial paralysis recovery or in the process of recovery, and many people have some understanding of its basic treatment, if not timely treatment of complications, in order to avoid such a situation, let's talk about how facial spasm is going on?.

How is facial spasm to return a responsibility?

First, hemifacial spasm is caused by pathological interference of facial nerve conduction caused by certain compression. Most patients with hemifacial spasm are caused by normal cross compression of blood vessels. The causes of hemifacial spasm include compression of posterior inferior cerebellar artery, anterior inferior cerebellar artery and vertebral nerve artery, and even compression of facial nerve root such as aneurysm, arteriovenous malformation or brain tumor.

Second: another part of patients with idiopathic facial paralysis, recovery after the secondary side muscle spasm. The cause of hemifacial spasm may be the pathological changes of demyelination caused by facial neuritis. There is still partial demyelination, which makes the electric conduction of facial nerve easy to be caused by generalization, or facial neuritis involves the nerve nuclei in the brain stem, forming epileptic lesions and producing paroxysmal twitch of facial muscles.

Third: a very small number of patients with traumatic tumor or surgical operation after the onset of lateral muscle spasm. It may be that there is a short circuit between facial nerve and other brain nerves during the recovery of facial nerve. When other nerves are excited, one side of facial muscle twitches. Hemifacial spasm is caused by deficiency of Yin in the body or weakness of Qi, deficiency of Yin, lack of blood, dystrophy of muscles and veins, or disturbance of wind and cold on the face; The nature of the disease is deficiency or excess. This is one of the causes of facial spasm.

matters needing attention

The typical manifestation of hemifacial spasm is intermittent twitch of orbicularis oculi muscle, which gradually extends to orbicularis oris muscle and facial expression muscle. In severe cases, mild facial pain can be felt when twitching. Mild convulsions can be stopped or reduced during sleep, and severe convulsions can not be stopped during sleep. Severe and chronic patients may have mild or severe facial paralysis.