What symptom can triglyceride uric acid have
summary
The high level of uric acid in our body is due to the disorder of purine metabolism in our body. This disorder of purine metabolism may increase the level of uric acid in patients. Hyperuricemia is a metabolic disease. When the human body has a high uric acid disease, it is possible to induce gout, due to the usual diet structure is not reasonable. Patients with hyperuricemia usually need to eat more substances that can reduce the content of uric acid, such as some alkaline foods, diuretic foods and foods with high water content. There is usually need to eat more vegetables and fruits, control the purine content of high food. This disease is also one of the most distressing diseases. Next, I will talk about the symptoms of this disease, hoping to help you.
What symptom can triglyceride uric acid have
First of all, hyperuricemia has different stages and usually has different manifestations in different stages. Hyperuricemia may start without symptoms, which is called my symptom hyperuricemia. Patients are often through blood tests to find that the concentration of uric acid in the blood is very high, but this time generally will not appear arthritis, kidney stones and other symptoms.
Secondly: high uric acid may also lead to gouty arthritis. At this time, the thumb joint and knee joint of the elder sister will be red and swollen due to the existence of gout. At the same time, the joint of the patient will have fever and severe pain. These symptoms usually attack at midnight.
Finally: if the uric acid content in the body continues to rise, it will lead to acute hyperuricemic arthritis. This kind of arthritis caused by hyperuricemic acid is most likely to attack in autumn, and the local joints of patients will be damaged, which may be caused by overeating or fatigue.
matters needing attention
When the body's uric acid content is too high, it may reach a saturation state. At this time, the uric acid in the body will form needle like crystal precipitation, which is generally in the joint synovial fluid of patients, forming gout.