chronic atrophic gastritis

Update Date: 2024-02-18 Source: Network

summary

At the beginning, I had stomachache and diarrhea at night (only once). I couldn't eat anything in the morning. I felt comfortable when I ate. Moreover, I felt better when I had diarrhea and bowed down. The doctor said that it was acute gastritis, which was much better after treatment. I'd like to share my experience in treating chronic atrophic gastritis with you

chronic atrophic gastritis

Treatment 1: general treatment: Patients with chronic atrophic gastritis, no matter what the cause, should quit smoking and avoid alcohol, avoid using drugs that damage the gastric mucosa, such as aspirin, indomethacin, erythromycin, etc., should eat regularly, avoid overheating, salty and spicy food, and actively treat chronic mouth, nose and throat infections.

Treatment 2: weak acid treatment: Patients with low acid or no acid confirmed by pentagastrin test can take appropriate amount of rice vinegar, 1-2 teaspoons each time, 3 times a day; or 10xff05; dilute hydrochloric acid, 0.5-10 ml, taken before or during meals, and pepsin mixture, 10 ml each time, 3 times a day; or multi enzyme tablets (DPP) or pancreatin tablets can be used to improve the symptoms of dyspepsia.

Treatment 3: anti Helicobacter pylori treatment: in chronic atrophic gastritis, the gastric acid is reduced or lacking, and the bacteria in the stomach are abnormal, especially the positive rate of Helicobacter pylori is very high. The application of antibiotics can improve the symptoms of chronic atrophic gastritis.

matters needing attention

I would also like to emphasize that too cold food and drinks can cause gastric spasm and vasoconstriction of gastric mucosa after cannibalism, which is not conducive to the regression of inflammation; overheated food and drinks will directly scald or stimulate gastric mucosa after eating. Gastritis patient's food should be soft and hard moderate, too hard coarse food, crude fiber vegetables, fried with oil or barbecue food, after eating can increase the burden of gastric mechanical digestion, so that the gastric mucosa by friction and damage, aggravate the mucosal inflammatory lesions.