What symptom does liver ascites have

summary

Cirrhotic ascites is commonly known as hepatoascites. There is a small amount of free ascites in the abdominal cavity of normal people, generally about 50 ml, which plays a role in maintaining the lubrication between organs. When there is too much free fluid in the abdominal cavity, it is called ascites. Ascites due to cirrhosis is a chronic liver disease. Let's take a look at the following.

What symptom does liver ascites have

First, by the massive, nodular, diffuse type of hepatocyte degeneration, necrosis, regeneration; regeneration, necrosis, promote tissue fiber hyperplasia and scar contraction, resulting in the hardening of the liver, the formation of cirrhosis. Portal hypertension is caused by liver dysfunction in cirrhosis, which leads to splenomegaly. Ascites is caused by the leakage of protein due to non absorption of protein and vitamins.

Second, excessive production of lymph: when the hepatic venous return is blocked, blood will penetrate from the wall of hepatic sinuses to the paranasal space, resulting in increased production of biliary lymph (about 7-11l per day, normal for 1-3l), which exceeds the capacity of thoracic duct drainage, and the lymph will seep from the hepatic capsule and hilar lymphatic vessels to the abdominal cavity.

Third: portal hypertension: hepatic portal vein and inferior vena cava, also known as the portal vein, it is the junction of the liver and other parts of the blood circulation. It is also the only way for hepatic artery and hepatic vein to enter and exit. Under normal conditions, the volume of the arteriovenous bed is basically the same, and the input and output blood flow are in balance.

matters needing attention

In cirrhosis, due to the degeneration and necrosis of hepatocytes and the proliferation of fibrous tissue, the vascular bed in the liver is compressed, twisted, deformed and narrowed, blocking the blood vessels, causing congestion in the hepatic sinuses, greatly reducing the blood flow, and the input volume is significantly greater than the output volume, which increases the pressure of the portal vein. At the same time, the capillary venous pressure also increased, over time, the gastrointestinal tract, mesentery, peritoneum and other blood reflux blocked, vascular permeability increased, the plasma components in the blood leaked, forming ascites.