Infantile facial neuritis symptoms?
summary
Infantile facial neuritis (Bell's palsy, Bell's palsy, idiopathic facial paralysis, idiopathic facial paralysis, primary facial paralysis, primary facial paralysis, primary facial paralysis, spontaneous facial paralysis, spontaneous facial paralysis, spontaneous facial paralysis), infantile facial neuritis symptoms? Let's talk about it.
Infantile facial neuritis symptoms?
In the upper group, facial paralysis resulted in the disappearance of frontal lines on the diseased side, inability to raise the forehead and frown, inability to close the eyelids or incomplete closure, and the eyeball turned upward to expose the white sclera when closing the eyes (known as Bell phenomenon). Because of orbicularis oculi muscle paralysis, lower eyelid ectropion, tear is not easy to flow into nasolacrimal duct and extraocular exudation. In the lower group, the symptoms of facial paralysis were shallow nasolabial sulcus, drooping mouth angle, mouth being pulled to the opposite side of the lesion, unable to pout and whistle, and air leakage at the mouth angle of the diseased side when blowing the cheek. Because of the paralysis of buccal muscles, it is easy to bite the buccal mucosa when chewing, and food is often retained between the teeth and cheek.
Severe injury, facial paralysis significantly, even in the face at rest. The lower part of the facial muscle of the patient was relaxed and the facial lines disappeared. The fissure of platysma was wider than normal. The random and cooperative movement of the facial muscle and platysma disappeared completely. When the patient tries to smile, the lower half of the facial muscle is pulled to the opposite side, resulting in the false appearance of deviation when the tongue is extended or the mouth is opened. Saliva and food gathered on the paralyzed side, and the patient could not close his eyes. When the lesion is located from peripheral nerve to ganglion, lacrimal gland nerve loses its function and cannot press tears into nasolacrimal duct through eyelid movement, resulting in excessive accumulation of tears in conjunctival sac. Because of the paralysis of the upper eyelid, the corneal reflex disappeared. The afferent part of corneal sensation and corneal reflex was indicated by blinking the other eyelid.
Partial facial nerve injury causes weakness in the upper and lower face, and occasionally the lower face is more affected than the upper face. The contralateral side was rarely involved. The recovery of facial paralysis depends on the severity of the disease. If the nerve has been cut off, the chance of complete or even partial recovery is very small. Most patients with facial paralysis can partially or completely recover their function. There is no difference in facial expression between the two sides when they are completely recovered at rest or during exercise; In partial recovery patients, there was "contracture" on the paralyzed side. Surface examination seemed to show that the normal side was weak. With the patient smiling or trying to move the facial muscle, this incorrect impression was more obvious.
matters needing attention
1. Strengthen the physique, pay attention to prevent the face from cold wind and upper respiratory tract infection. 2. Early comprehensive treatment to reduce complications and sequelae.