Where is sensory aphasia bad?

summary

Aphasia refers to the pathological changes of brain tissue related to language function, which brings great harm to patients, such as stroke, brain trauma, brain tumor, brain inflammation, etc., resulting in the impairment of patients' understanding and expression ability of human communication symbol system. Now let's talk about what's wrong with sensory aphasia?.

Where is sensory aphasia bad?

First: hysterical aphasia, also known as aphasia or functional aphasia, is a temporary voice disorder caused by obvious psychological factors, which is more common. Clinical manifestations: sudden loss of his voice, or just a whisper, tears and laughter or cough are usually normal. Laryngoscope can see vocal cords in the booth, and deep breathing is even more laughing or coughing.

Second, motor aphasia, also known as expressive aphasia, Broca aphasia, and non fluent aphasia, is caused by lesions in the tegmentum and trigonometric cortex at the back end of the inferior frontal gyrus, that is, the speech motor center. The patient can not speak at all, or can only speak 1-2 simple words without fluency, and can not use words properly, but he can understand other people's words and know his own wrong words; I can understand what I write, but I have difficulties and mistakes in reading, and most of them are accompanied by hemiplegia.

Third, sensory aphasia is also called Wernicke aphasia, receptive aphasia and posterior aphasia. The lesion was located in the superior temporal gyrus of the dominant hemisphere, i.e. the left Wernicke area and the adjacent inferior parietal cortex, and supplied by the lower branch of the left middle cerebral artery.

matters needing attention

1. Transient ischemic attacks (TIA): the onset age is more than 50 years old, and most of them have a history of arteriosclerosis. It usually lasts for several minutes to several hours, and generally recovers completely within 24 hours. It can attack repeatedly and intermittently without neurological symptoms. It may be accompanied by unilateral blindness, hemiplegia, hemisensory disturbance and other neurological symptoms and signs. It is often caused by microembolization of atherosclerotic plaque, cerebral arteriospasm, cardiac insufficiency and acute hypotension. 2. cerebral thrombasis: the age of onset is higher, and the incidence rate is higher than 60 years old. There are more history of hypertension, diabetes, arteriosclerosis and other organ sclerosis. The disease is more common in quiet time, and the symptoms are often found after waking up in the morning. The symptoms usually worsen gradually in a few hours or a long time, showing trapezoidal progression. The consciousness remained clear and there were focal neurological dysfunction such as hemiplegia. 6 hours after the onset, cerebrospinal fluid generally did not contain blood. Cerebral angiography and CT are helpful to the final diagnosis.