Can vitiligo eat sweet potato

Update Date: Source: Network

summary

Vitiligo disease has been half a month, eyelids, armpits have white spots, the area of corn kernels, treatment for many times, just began to have effect, the color of white spots have a trend of becoming lighter, but the more to the back, the effect is not obvious, after the treatment of expert system, now the situation is also stable, so vitiligo can eat sweet potato, you know? Let's talk about whether vitiligo can eat sweet potato.

Can vitiligo eat sweet potato

Food one: Patients with vitiligo is not suitable to eat spinach. Although spinach is rich in vitamins, it also contains oxalic acid, which can interact with chemicals in human blood. Therefore, spinach is one of the factors that hinder the treatment, aggravate the disease, expand the symptoms and make the disease not heal for a long time. Spinach should never be eaten in life.

Food 2: vegetables rich in vitamin C are also the majority of vitiligo patients should try to eat less or not to eat, such as tomatoes, hawthorn, red bayberry, cherry, orange, orange, grapefruit and so on. These fruits are rich in vitamin C and have depigmentation effect on skin. In the process of melanin metabolism, they can interrupt the production of melanin and aggravate the condition of patients with vitiligo. On the contrary, it is not conducive to the rehabilitation and treatment of vitiligo.

Food three: vitiligo can not eat vegetables and cabbage, rape, garlic, chrysanthemum, coriander, amaranth, mustard, lotus root, green pepper, cauliflower, balsam pear, shepherd's purse, Toona sinensis, leek, fennel, mustard, cabbage, garlic, pepper, pea seedlings, chrysanthemum brain, mustard, alfalfa, cabbage and so on. Many vegetables are not suitable for eating.

matters needing attention

Warm tips: for the lack of elements is the main cause of vitiligo, especially some metal elements, such as copper, iron, zinc. Clinical studies have found that juvenile vitiligo is mainly due to the lack of trace elements in order to lead to the lack of tyrosinase activity in the body of patients, melanin cell damage, and then reduce melanin synthesis.