Is there a cure for adenomatous gastric polyps

Update Date: Source: Network

summary

Bird's nest is good for skin, but not for stomach. Because bird's nest is collagen, the more people with stomach distension eat, the more difficult it is to digest, and the gelatinous things are more difficult to digest. As a more complex disease, can we cure adenomatous gastric polyps?

Is there a cure for adenomatous gastric polyps

Treatment 1: general treatment: Patients with chronic atrophic gastritis, no matter what the cause, should quit smoking and avoid alcohol, avoid using drugs that damage the gastric mucosa, such as aspirin, indomethacin, erythromycin, etc., should eat regularly, avoid overheating, salty and spicy food, and actively treat chronic mouth, nose and throat infections.

Treatment 2: weak acid treatment: Patients with low acid or no acid confirmed by pentagastrin test can take appropriate amount of rice vinegar, 1-2 teaspoons each time, 3 times a day; or 10xff05; dilute hydrochloric acid, 0.5-10 ml, taken before or during meals, and pepsin mixture, 10 ml each time, 3 times a day; or multi enzyme tablets (DPP) or pancreatin tablets can be used to improve the symptoms of dyspepsia.

Treatment 3: anti Helicobacter pylori treatment: in chronic atrophic gastritis, the gastric acid is reduced or lacking, and the bacteria in the stomach are abnormal, especially the positive rate of Helicobacter pylori is very high. The application of antibiotics can improve the symptoms of chronic atrophic gastritis.

matters needing attention

This disease needs attention: too cold food and drink can cause gastric spasm and vasoconstriction of gastric mucosa after cannibalism, which is not conducive to the regression of inflammation; overheated food and drink will directly scald or stimulate gastric mucosa after eating. Gastritis patient's food should be soft and hard moderate, too hard coarse food, crude fiber vegetables, fried with oil or barbecue food, after eating can increase the burden of gastric mechanical digestion, so that the gastric mucosa by friction and damage, aggravate the mucosal inflammatory lesions.