How to treat corneal vascular hyperplasia
summary
Neovascular glaucoma is not caused by intraocular hemorrhage, so although it can be explained in medicine, the treatment is relatively difficult. Patients with neovascular glaucoma usually feel eye pain, photophobia, poor vision, can reach the index of things in front of their eyes, high intraocular pressure, can reach more than 60mmhg, and even severe congestion. Now I'll tell you about it.
How to treat corneal vascular hyperplasia
First: the drug treatment of neovascular glaucoma (NVG): local combined application of 1% atropine, twice a day and glucocorticoid, four times a day, even if the intraocular pressure is as high as 60mmhg, most patients can fully relieve the symptoms. If there is microcystic edema in the cornea, the above drugs combined with soft contact lenses can usually work. However, it should be noted that long-term use of this treatment is not without complications. Even in advanced glaucoma, it is necessary to reduce intraocular pressure and prevent chronic corneal decompensation.
Second: the drug treatment of iris neovascularization (NVI): in the case of open angle, conventional anti glaucoma drugs can also effectively reduce intraocular pressure, but unless given PrP treatment to prevent angle closure, the effect of drug treatment is only temporary relief. During this period, local application of 1% atropine, twice a day to relieve ocular congestion, glucocorticoid 4 times a day to relieve ocular inflammation.
Third: neovascular glaucoma (NVG) surgical treatment: active neovascularization through the filtering site, resulting in conjunctival scar formation, leading to the failure of the follicle. Before the extensive application of PrP, various surgical treatments, such as electric cauterization iridotomy, diathermy iridotomy, bipolar micro cauterization, non penetrating ciliary body diathermy, and even upper iridectomy to eliminate the source of bleeding, were used to control intraoperative bleeding.
matters needing attention
Because the operation under high intraocular pressure can cause many complications, therefore, strengthening the treatment of intraocular pressure before operation, reducing the intraocular pressure as much as possible, and combining the application of hemostasis is the key to facilitate the operation, avoid intraoperative neovascularization bleeding, and improve the cure rate. At the same time, the patients and their families should be aware of the healing process of the disease, emphasize the possible complications and eye discomfort within one week after the operation, and relieve the unnecessary panic and doubts of the patients in the future.