What examination does gastric mucous membrane thicken do?

summary

Gastric mucosa thickening is one of the clinical manifestations of "chronic atrophic gastritis" (intestinal metaplasia, atypical hyperplasia). Chronic atrophic gastritis is a common gastric disease. Arteriosclerosis, insufficient gastric blood flow, tobacco, alcohol and tea are easy to damage the barrier function of gastric mucosa and cause chronic atrophic gastritis. What examination does gastric mucous membrane thicken do? Let's talk about it

What examination does gastric mucous membrane thicken do?

Pepsinogen determination: pepsinogen is secreted by the main cell. In chronic atrophic gastritis, the content of pepsinogen in blood and urine decreases. Immunological examination: Determination of parietal cell antibody (PCA), internal factor antibody (IFA) and gastrin secreting cell antibody (GCA) can be used as auxiliary diagnosis of chronic atrophic gastritis and its classification.

Determination of serum gastrin: G cells in antral mucosa secreted gastrin. In patients with type a CAG, the serum gastrin level was increased significantly; The atrophy of antral mucosa in patients with type B CAG directly affects the secretion of gastrin by G cells, and the serum gastrin level is lower than normal.

X-ray examination: X-ray barium meal examination showed no abnormality in most patients with atrophic gastritis. The results of gas barium double contrast examination showed that the folds of gastric body mucosa were flat and thin, the serrated folds of gastric body were thin or disappeared, the bottom of the stomach was smooth, and some of the gastric sinusitis mucosa were serrated or rough.

matters needing attention

The food should be fresh and nutritious to ensure adequate intake of protein, vitamins and iron. Eat on time, don't overeat, don't eat too cold or too hot food, don't use or less pungent condiments such as spicy powder. In order to avoid the damage of nicotine to gastric mucosa, drinking alcohol and smoking should be controlled; Avoid long-term use of anti-inflammatory analgesics, such as aspirin and corticosteroids, in order to reduce gastric mucosal damage.