How does uterine Apoplexy Syndrome form?

summary

Uterine Apoplexy Syndrome is also known as senile endometrial hemorrhagic necrosis. After menopause, the patient developed endometrial necrosis, vaginal bleeding and cardiac dysfunction. Postmenopausal vaginal bleeding, dark mass, longer course. With palpitations, shortness of breath, cough, shortness of breath, chest tightness and pain, or liver swelling, physical weakness. So how does uterine Apoplexy Syndrome form?

How does uterine Apoplexy Syndrome form?

With the increase of age, different degrees of arteriosclerosis occur, and the gradual decline of ovarian function leads to the decrease of estrogen secretion, which further promotes and aggravates the arteriosclerosis. This is the manifestation of uterine arteriosclerosis. The syndrome is more common in postmenopausal women. Abnormal vaginal bleeding caused by arteriosclerosis, uterine artery insufficiency, endometrial necrosis or shedding; Intrauterine infection caused by decreased resistance, local long-term abnormal bleeding or blood loss; The symptoms and signs of ischemic heart disease were different;

Uterine stroke syndrome is not inherited. Diseases can be divided into genetic diseases and non genetic diseases. Genetic disease refers to the disease which is completely or partly determined by genetic factors. It is often congenital and can also be acquired. Those caused by infection or injury are non genetic diseases. Uterine Apoplexy Syndrome is a non hereditary disease, and its etiology is as follows: with the increase of age, different degrees of arteriosclerosis occur, and the gradual decline of ovarian function leads to the decrease of estrogen secretion, which promotes and aggravates the arteriosclerosis. This is the manifestation of uterine arteriosclerosis.

If after 20 weeks of gestation or delivery period, the normal placental position before the delivery of the fetus, part or all of the bleeding phenomenon from the uterine wall, known as placental abruption. When internal hemorrhage occurs in placental abruption, blood accumulates between placenta and uterine wall, causing hematoma. With the increase of hematoma pressure after placenta, blood immerses into uterine myometrium, causing separation, rupture and even degeneration of muscle fibers. When blood penetrates into uterine serosa, the surface of uterus presents purple blue ecchymosis, which is called uterine apoplexy.

matters needing attention

The treatment of this disease is mainly aimed at the cause of disease, such as the improvement of systemic vascular state and medication for arteriosclerosis; In case of infection, antibiotics should be used timely and effectively; Some hemostatic agents can be used when bleeding. Postmenopausal women (especially postmenopausal women after 45 years old) had better do blood lipid analysis and hemorheology examination once a year. Once cholesterol, triglyceride and blood viscosity increase, effective measures should be taken in time to strengthen physical exercise and eat less fatty food.