Food for chronic gastritis

summary

Patients with chronic gastritis should eat a regular and quantitative diet, and eat more easily digestible food. Eat less and eat more. The key to the treatment of stomach disease is to develop a good eating habit. Today, let me talk about the food for treating chronic gastritis.

Food for chronic gastritis

First: porridge food is absolutely the best choice, especially millet porridge has obvious effect of stomach. However, not all people are suitable for eating porridge to nourish their stomach. In the past, porridge was cooked for a long time, so it would decompose the rice grains into disaccharides or monosaccharides. Carbohydrates are the most likely to stimulate the secretion of gastric acid, and some people also have acid vomiting.

Second: in the gastritis eat what food is good, soup is also essential, in fact, the more stomach nourishing things preferred is not porridge, but soup, and soup can not be broth. This is because the broth contains a lot of acidic substances, the gastric mucosa will have a certain stimulating effect, so it is best to drink more vegetable soup. For example, spinach, vermicelli and eggs can be served, and chicken essence can be added to flavor, which is not only delicious, but also very nourishing.

Third: in terms of staple food, the most stomach nourishing food is noodles. In the rice we often eat, there are certain acidic substances, so people with bad stomach should eat less rice. If it is porridge, you can put some baking soda in it, which has a certain maintenance effect on the stomach. In addition to noodles, another stomach food is soda biscuits.

matters needing attention

I would also like to emphasize that anti parietal cell antibodies can be found in the blood and gastric juice of some patients with atrophic gastritis, and there is diffuse lymphocyte infiltration in the gastric mucosa. It is believed that the autoimmune reaction may be related to the occurrence of atrophic gastritis. The pathological changes of superficial gastritis were congestion, edema, exudation, erosion and bleeding of gastric mucosa; atrophic gastritis was flat or disappeared of mucosal folds; hypertrophic gastritis was thickening of mucosa and hyperplasia of epithelial cells. The main clinical manifestations are dyspepsia, epigastric pain after eating, anemia and upper gastrointestinal bleeding.