Is aortopulmonary artery dilation serious?

summary

Is aortopulmonary artery dilation serious? Pulmonary artery dilatation refers to the enlargement of the common trunk of the pulmonary artery without other malformations, which may be caused by the uneven differentiation of the arterial trunk during embryonic development, resulting in the larger pulmonary artery and smaller aorta. Most patients have no symptoms or functional symptoms. Physical examination showed that the boundary of heart murmur was not enlarged, and there were below grade 3 systolic blowing murmur and systolic ejection sound in the pulmonary valve area, and the second heart sound split and slightly hyperactivity.

Is aortopulmonary artery dilation serious?

X-ray showed that the pulmonary artery segment was protruding, the heart shadow was not big, and the pulmonary vascular shadow was normal. ECG was normal, echocardiography showed pulmonary trunk widening, cardiac catheterization showed no abnormality, angiocardiography showed pulmonary trunk dilation. Diagnosis relies on the exclusion of congenital and acquired cardiovascular diseases that can cause pulmonary artery dilatation. The prognosis of this disease is good and no special treatment is needed.

Most patients have no symptoms or functional symptoms. Physical examination showed that the boundary of heart murmur was not enlarged, and there were below grade 3 systolic blowing murmur and systolic ejection sound in the pulmonary valve area, and the second heart sound split and slightly hyperactivity. X-ray showed that the pulmonary artery segment was protruding, the heart shadow was not big, and the pulmonary vascular shadow was normal. ECG was normal, echocardiography showed pulmonary trunk widening, cardiac catheterization showed no abnormality, angiocardiography showed pulmonary trunk dilation.

Pulmonary artery dilatation refers to the enlargement of the common trunk of the pulmonary artery without other malformations, which may be caused by the uneven differentiation of the arterial trunk during embryonic development, resulting in the larger pulmonary artery and smaller aorta. Most patients have no symptoms or functional symptoms. Physical examination showed that the boundary of heart murmur was not enlarged, and there were below grade 3 systolic blowing murmur and systolic ejection sound in the pulmonary valve area, and the second heart sound split and slightly hyperactivity.

matters needing attention

The diet of patients should cooperate with relevant clinical treatment to help the heart work stably, maintain reasonable weight, reduce edema, and provide necessary nutritional support for the body. There is no such diet for all patients, there is no so-called "diet standard".