Is left ventricular dysplasia serious?

summary

Hypoplastic left heart is a rare and complex congenital cardiovascular malformation. The incidence rate is about 1 ~ 27/10 million, accounting for about 1.4% of congenital cardiovascular malformations. The common point of this kind of malformation is that there is stenosis or atresia in the left cardiac circulation, which makes the left atrium, pulmonary vein and pulmonary artery dilate and increase the pressure, so the blood flow of the right ventricle increases, and it must be accompanied by an arterial catheter to survive. Let's share my experience with you.

Is left ventricular dysplasia serious?

The left ventricular diastolic phase of patients with aortic regurgitation received both left atrial blood and additional blood from the aorta, resulting in the gradual increase of left ventricular end diastolic volume. With the compensation mechanism of myofibrillar segment, the left ventricle produces eccentric left ventricular hypertrophy and the left ventricular compliance increases, so as to gradually adapt to the chronic left ventricular volume overload, Guarantee - left ventricular end diastolic volume increases while left ventricular end diastolic pressure is in the normal range,

This mechanism of myocardial compensation can be maintained for a long time, making the patient asymptomatic. However, with the progress of the disease, the left heart is mainly in the subendocardial region. With the decline of left ventricular systolic and diastolic function, the symptoms appear, and soon the decline of left ventricular function is irreversible; Therefore, due to the mechanism of myocardial compensation, these patients often fail to see a doctor in time for a long period of time because they have no symptoms. Once the symptoms appear, they will soon cause irreversible changes in left ventricular function. Even after surgery, the prognosis is relatively poor.

About 3% of patients with coronary atherosclerotic heart disease have mitral regurgitation by cardiac catheterization. Mitral regurgitation caused by coronary heart disease can be caused by acute or chronic papillary muscle ischemia. In myocardial infarction, papillary muscle can be completely ruptured within a few hours due to acute ischemic necrosis, although chordae tendineae and leaflets have no abnormal lesions, However, the mitral valve at the corresponding position lost its opening and closing function,

matters needing attention

This disease is often accompanied by acidosis, hypoglycemia, hypoxemia and shock. Cyanosis may not be obvious if the flow distribution of atrial septal defect is large, and hypoxemia is mild. About 90% of the children often die within one month after birth if they are not diagnosed and treated in time.