Does second liver make water yellow astringent symptom?

summary

Liver disease is the major culprit of strangling people's health in the 21st century, and a series of complications brought by liver disease can not be ignored. If liver disease is not treated in time, it will cause bad consequences and pose a great threat to our lives. Next to introduce the symptoms of hepatitis B urine yellow astringent?

Does second liver make water yellow astringent symptom?

（1） Hepatitis A virus (HAV) is a kind of RNA virus, which belongs to the family micrornaviridae. It is a spherical particle with a diameter of about 27 nm. It consists of 32 shell particles and a symmetrical 20 hedral nucleocapsid, containing linear single strand RNA. Hav has four main polypeptides, namely VP1, VP2, VP3 and VP4. VP1 and VP3 are the main antigenic polypeptides of viral capsid protein, which induce neutralizing antibodies. Hav has strong resistance in vitro, and its infectivity remains unchanged after being stored at - 20 ℃ for several years. It can tolerate the temperature of 56 ℃ for 30 minutes and the acidity of PH3;

（2） Hepatitis B virus (HBV) is a kind of DNA virus, belonging to hepadnavidae, which is a spherical particle with a diameter of 42 nm. Also known as Dane particles, it has a shell and a core. The shell is 7-8nm thick, with HBsAg and core diameter of 27nm. It contains partial double stranded and partial single stranded circular DNA, DNA polymerase, core antigen and e antigen. The genome of HBV DNA contains about 3200 base pairs. The length of the long chain was fixed, and there was a nick, where was Dan polymerase; The length of the short chain is variable. When HVB replicates, endogenous DNA polymerase repairs the short strand to form a complete double strand structure, and then transcribes it. There are four open reading frames (ORFs) in the long chain of HBV DNA: s, C, P and X.

Hepatitis B e antigen (HBeAg) and e antibody - (HBE) HBeAg are soluble proteins in the core of hepatitis B virus (HBV) in a covert form. Their coding genes overlap with each other and are the sub components of HBcAg. After HBV infection, HBeAg and HBsAg can appear in the blood at the same time or later, and disappear earlier than HBsAg. HBsAg only exists in the blood of HBsAg positive patients, usually accompanied by replication of HBV DNA in the liver. There are many Dane particles and increased activity of HBV DNA polymerase in the blood. Therefore, HBeAg positive is an important index of active replication of the virus, which is highly infectious.

matters needing attention

Due to the destruction of liver cells caused by hepatitis virus, which affects the metabolism of bilirubin, bilirubin enters the blood and is discharged from the body through urine, which is more than usual, so the color of urine is deepened. The more yellow the color of urine, the more serious the destruction of liver cells, the better the condition, the urine color gradually returned to normal.