How does salivary gland virus disease get?

summary

Salivary adenovirus disease is also called salivary gland giant cell inclusion body disease. It mostly occurs in infants under 2 years old, and it can also occur in adults, but it is rare. Fetal growth retardation caused by transplacental infection can lead to premature delivery. How does salivary gland virus disease get?

How does salivary gland virus disease get?

Clinically, there are obvious hepatosplenomegaly, fever, jaundice, thrombocytopenic purpura and nervous system invasion. It has been reported that 10% - 30% of the salivary glands of stillborn infants have the manifestation of inclusion body disease. Adult onset is related to extremely weak constitution, and abnormal immune response will appear during infection.

The disease is caused by cytomegalovirus infection. The main route of infection is contact infection, and infants can be infected through placenta. It is more common in submandibular gland and parotid gland. Characteristic cells can be found in saliva, sputum and urine. There is no specific treatment for this disease, only according to the clinical symptoms of patients with symptomatic treatment.

Chronic type is more common than acute type, accounting for about 80%, mostly in adults. The course of the disease is slow, and the main bleeding symptom is skin purpura with more limbs, followed by nasal gingival and oral mucosal bleeding. The general bleeding symptoms are mild and extensive, while severe bleeding is rare. Thrombocytopenia can be seen in the blood examination, generally in 50 × The treatment of chronic type is similar to that of acute type, but the effect of hormone is not as good as that of acute type. If necessary, splenectomy can be considered.

matters needing attention

This disease should seek medical treatment in time. Severe cases in the upper acute stage should be absolutely bedridden to prevent severe bleeding. The purpose of treatment includes: controlling bleeding symptoms, reducing platelet damage and increasing the number of platelets. Hormone is the first choice for treatment. Prednisone can be taken orally. Hydrocortisone can be given intravenously when the patient is in critical condition. Hemostatic agents such as anluoxue, zhizhimin anti fibrinolytic aromatic acid and 6-aminocaproic acid were used. Fresh blood could be transfused to patients with severe bleeding. Platelet transfusion is also needed when conditions permit.