Type 3 symptoms of aortic dissection?
summary
Aortic dissection (AD) refers to the separation of true and false lumens of the aortic wall, in which the blood in the aortic lumen enters the aortic media from the tear of aortic intima to separate the media and expand along the long axis of the aorta. The disease is rare, with an incidence rate of about 5~10 per million people per year, with a peak age of 50~70 years, and a male: female: about 2~3: 1. 65% ~ 70% of them died of cardiac tamponade and arrhythmia in the acute stage, so early diagnosis and treatment are very necessary. Type 3 symptoms of aortic dissection? Let's talk about it
Type 3 symptoms of aortic dissection?
Most patients have sudden chest and back pain. Type A is more common in the anterior chest and interscapular area, while type B is more common in the back and abdomen. The pain was severe and intolerable, reaching the peak after the onset of the disease, presenting as knife cutting or tearing. The pain of a few patients with slow onset was not significant.
Aortic regurgitation occurs when dissection hematoma involves aortic valve ring or affects the support of valve leaflet. Diastolic whistling murmur, pulse pressure widening and acute aortic regurgitation may suddenly appear in aortic valve area, which may cause heart failure. The change of pulse pressure is usually seen in the stiff, brachial or femoral arteries. One side of the pulse is weakened or disappeared. The origin of the branch of the anti aorta is compressed or blocked by intimal lobes. Can have pericardial rub fricative, pleural effusion.
Most patients may have hypertension. Patients with severe pain and shock appearance, anxiety, sweating, pale, heart rate acceleration, but blood pressure is often not low or even increased. When the dissection involves the blood supply of visceral artery, limb artery and spinal cord, the corresponding organ tissue ischemia, renal ischemia, lower limb ischemia or paraplegia and other neurological symptoms may appear.
matters needing attention
Combination of blood pressure control and heart rate reduction β Receptor blockers and vasodilators were used to reduce vascular resistance, vascular wall tension and ventricular contractility, DP / dt of left ventricle, and blood pressure was controlled at 100-120 mmHg. The heart rate is between 60 and 75 beats / min to prevent the expansion of the lesion.