How does huge bladder do?
summary
Giant bladder giant ureter syndrome, also known as giant bladder complicated with giant ureter syndrome, is characterized by huge thickening of bladder ureter, but no lower urinary tract obstruction, vesicoureteral reflux, and no lower urinary tract obstruction. Some people think that giant bladder or giant ureter should be diagnosed as their own independent diseases. What about giant bladder?
How does huge bladder do?
The cause of huge bladder. The results are as follows: 1. The number of parasympathetic ganglion cells in the wall of the distal ureter is decreased, and the parasympathetic ganglion cells in the wall of the distal ureter are decreased, hypoplastic or absent, resulting in the loss of peristaltic function of the ureter and functional obstruction, but it has not been confirmed by histology. 2. The lack of longitudinal muscle in the wall of the distal ureter (normal ring muscle) results in functional obstruction, It is suggested that the deficiency of longitudinal muscle is due to the compression of ureter by mesonephric tube after 12 weeks of embryo
According to the preventive measures of birth defects, prevention should run from pre pregnancy to prenatal. Premarital physical examination plays a positive role in the prevention of birth defects, and the role depends on the examination items and contents.
Differential diagnosis of giant bladder. 1. Obstructive megaureter, congenital ureteral stenosis, valve, atresia, ectopic opening, acquired ureteral polyps, stones, infection, trauma, etc. can cause obstructive megaureter. 2. Reflux megaureter, primary or secondary vesicoureteral reflux, such as secondary to congenital posterior urethral valve, urethral stricture, urethral diverticulum, neurogenic bladder It is difficult to differentiate neurogenic bladder from the disease
matters needing attention
Giant bladder examination. According to the clinical recurrent urinary tract infection, combined with intravenous secretory radiography and cystography can make a preliminary diagnosis; ② Bilateral ureteral dilatation; ③ Bladder distention; ④ Irreversibility; ⑤ There was no lower urinary tract obstruction; ⑥ No neurogenic bladder was found; ⑦ No dysuria